Neuroscience & regenerative medicine


Bohl D, Pochet R, Mitrecic D, Nicaise C. Modelling and treating amyotrophic lateral sclerosis through induced-pluripotent stem cells technology. Curr Stem Cell Res Ther. 2016;11(4):301-12.


Pochet R, Nicaise C, Mitrečić D. Translation of the focus toward excellence in translational science: comment on "TDP-43 Repression of Nonconserved Cryptic Exons is Compromised in ALS-FTD". Croat Med J. 2015 Oct 31;56(5):493-5.

Nicaise C, Mitrecic D, Falnikar A, Lepore AC. Transplantation of stem cell-derived astrocytes for the treatment of amyotrophic lateral sclerosis and spinal cord injury. World J Stem Cells. 2015 Mar 26;7(2):380-98.

Zheng M, Zhang H, Dill DL, Clark JD, Tu S, Yablonovitch AL, Tan MH, Zhang R, Rujescu D, Wu M, Tessarollo L, Vieira W, Gottesman MM, Deng S, Eberlin LS, Zare RN, Billard JM, Gillet JP, Li JB, Peltz G. The role of Abcb5 alleles in susceptibility to haloperidol-induced toxicity in mice and humans. PLoS Med. 2015 Feb 3;12(2):e1001782.


Li K, Nicaise C, Sannie D, Hala TJ, Javed E, Parker JL, Putatunda R, Regan KA, Suain V, Brion JP, Rhoderick F, Wright MC, Poulsen DJ, Lepore AC. Overexpression of the astrocyte glutamate transporter GLT1 exacerbates phrenic motor neuron degeneration, diaphragm compromise, and forelimb motor dysfunction following cervical contusion spinal cord injury. The Journal of neuroscience : the official journal of the Society for Neuroscience 2014, 34:7622-7638.

Allard J, Li K, Lopez XM, Blanchard S, Barbot P, Rorive S, Decaestecker C, Pochet R, Bohl D, Lepore AC, Salmon I, Nicaise C. Immunohistochemical toolkit for tracking and quantifying xenotransplanted human stem cells. Regen Med. 2014;9(4):437-52.


Wanet A, Tacheny A, Arnould T, Renard P miR-212/132 expression and functions: within and beyond the neuronal compartment. Nucleic acids research 2012, 40:4742-4753.a

Rommelaere G, Michel S, Malaisse J, Charlier S, Arnould T, Renard P Hypersensitivity of A8344G MERRF mutated cybrid cells to staurosporine-induced cell death is mediated by calcium-dependent activation of calpains The international journal of biochemistry & cell biology 2012, 44:139-149.

Dombret C, Nguyen T, Schakman O, Michaud JL, Hardin-Pouzet H, Bertrand MJ, De Backer O. Loss of Maged1 results in obesity, deficits of social interactions, impaired sexual behavior and severe alteration of mature oxytocin production in the hypothalamus. Hum Mol Genet. 2012 Nov 1;21(21):4703-17.